A recent study from Geisinger Medical Center has found that two types of drugs used to treat high blood pressure for the ...

Renal hypoperfusion occurs as a result of reduced cardiac output, resulting in the activation of the renin–angiotensin–aldosterone system, which compensates for the hypoperfusion. However ...

such as the renin-angiotensin-aldosterone system and the sympathetic nervous system. The activated neurohormones angiotensin II, aldosterone, and norepinephrine promote tubular sodium reabsorption ...

A new study that looked at how liberal vs. restrictive fluid restriction impacts health status for patients with chronic ...

Blood pressure begins to fall, and the angiotensin-aldosterone system is activated and causes an elevation in aldosterone. Elevated aldosterone stimulates sodium reabsorption in the cortical ...

physicians must be aware of clinical circumstances that may promote the development of hyperkalemia The renin–angiotensin–aldosterone system (RAAS) has a pathogenetic role in several edematous ...

The two primary mechanisms — considered “neurohormonal” responses — are activation of the sympathetic nervous system (SNS) and activation of the renin-angiotensin-aldosterone system (RAAS).

Lisinopril inhibits angiotensin-converting enzyme. It suppresses the renin-angiotensin-aldosterone system. The mechanism of the antihypertensive effect of thiazides is unknown. Fetal toxicity may ...

The dual-action agent LCZ696 blocks the angiotensin II receptor (via a valsartan moiety) and inhibits neprilysin (via an AHU377 moiety). In a phase II trial, this drug “demonstrated superior ...